In today's e-mail newsletter regarding the switch from cholesterol to inflammation as a target for statins, Dr.Graveline said, "Hopefully soon we will have an anti-inflammatory statin that does not block the mevalonate pathway. That will be true medical progress."
My question: Why does the anti-inflammatory element have to be a statin at all? I have used serrapeptase, an enzyme, for good effect in dealing with muscle-joint inflammation, including that caused by statins.
I have some concerns about serrapeptase because, although it is represented to be widely prescribed or recommended in Europe, especially Germany, by physicians, I have seen no studies (written in English) that could be considered to be reliable. Back in the old days (in the 1950's, when I was in high school), we were told that in order to become a physician, one had to study German, French, and Latin. I think those requirements have been dropped so today's physicians can't read the German studies. And neither can I.
Has anyone else used or prescribed serrapeptase for inflammation?